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Pubblicazioni
CANCER DISCOVERY
Rivista
Codice:
E210709
ISSN:
2159-8274
Dati Generali
Dati Generali
Pubblicazioni (35)
Pulisci
Ordina Pubblicazioni:
ascendente
decrescente
A genetic platform to model sarcomagenesis from primary adult mesenchymal stem cells
Articolo
A molecularly annotated platform of patient-derived xenografts (‘xenopatients’) identifies HER2 as an effective therapeutic target in cetuximab-resistant colorectal cancer
Articolo
Acquired resistance to the TRK inhibitor entrectinib in colorectal cancer
Articolo
Adaptive Evolution: How Bacteria and Cancer Cells Survive Stressful Conditions and Drug Treatment
Articolo
Amplification of the MET Receptor Drives Resistance to Anti-EGFR Therapies in Colorectal Cancer.
Articolo
Cancer Hallmarks: Piecing the Puzzle Together
Articolo
Cd4 t cell–dependent rejection of beta-2 microglobulin null mismatch repair–deficient tumors
Articolo
Cell-autonomous and non-cell-autonomous mechanisms of HGF/MET-driven resistance to targeted therapies: from basic research to a clinical perspective.
Articolo
Climbing RAS, the Everest of Oncogenes.
Articolo
Combining a PI3K inhibitor with a PARP inhibitor provides an effective therapy for BRCA1-related breast cancer.
Articolo
Compensatory pathways in oncogenic kinase signaling and resistance to targeted therapies: Six degrees of separation
Articolo
Comutations and KRASG12C Inhibitor Efficacy in Advanced NSCLC
Articolo
EGFR blockade reverts resistance to KRAS G12C inhibition in colorectal cancer
Articolo
Gene Signatures Associated with Mouse Postnatal Hindbrain Neural Stem Cells and Medulloblastoma Cancer Stem Cells Identify Novel Molecular Mediators and Predict Human Medulloblastoma Molecular Classification.
Articolo
HER2 Activating Mutations Are Targets for Colorectal Cancer Treatment
Articolo
In Vivo Role of INPP4B in Tumor and Metastasis Suppression through Regulation of PI3K-AKT Signaling at Endosomes
Articolo
MET-driven resistance to dual EGFR and BRAF blockade may be overcome by switching from EGFR to MET inhibition in BRAF mutated colorectal cancer
Articolo
Macrophage PI3Kγ drives pancreatic ductal adenocarcinoma progression
Articolo
Molecular heterogeneity and receptor coamplification drive resistance to targeted therapy in MET-Amplified esophagogastric cancer
Articolo
Oncogenic MET as an effective therapeutic target in non-small cell lung cancer resistant to EGFR inhibitors: The rise of the phoenix
Articolo
Patient-Derived Xenograft Models: An Emerging Platform for Translational Cancer Research.
Articolo
Polyclonal Secondary FGFR2 Mutations Drive Acquired Resistance to FGFR Inhibition in Patients with FGFR2 Fusion-Positive Cholangiocarcinoma
Articolo
Rational treatment of metastatic colorectal cancer: A reverse tale of men, mice, and culture dishes
Articolo
Resistance to Anti-EGFR Therapy in Colorectal Cancer: From Heterogeneity to Convergent Evolution.
Articolo
Suppression of CHK1 by ETS Family Members Promotes DNA Damage Response Bypass and Tumorigenesis
Articolo
TAS-120 overcomes resistance to atp-competitive FGFR inhibitors in patients with FGFR2 fusion–positive intrahepatic cholangiocarcinoma
Articolo
TIMP1 mediates astrocyte-dependent local immunosuppression in brain metastasis acting on infiltrating CD8+ T cells
Articolo
TRK xDFG mutations trigger a sensitivity switch from type I to II kinase inhibitors
Articolo
Temozolomide treatment alters mismatch repair and boosts mutational burden in tumor and blood of colorectal cancer patients
Articolo
The APL Paradigm and the "Co-Clinical Trial" Project
Articolo
The Clinical Impact of the Genomic Landscape of Mismatch Repair-Deficient Cancers
Articolo
The gRASs Is Greener: Potential New Therapies in Lung Cancer with Acquired Resistance to KRASG12C Inhibitors
Articolo
Tumor evolution as a therapeutic target
Articolo
Tumor heterogeneity and Lesion-Specific response to targeted therapy in colorectal cancer
Articolo
Werner helicase is a synthetic-lethal vulnerability in Mismatch Repair-Deficient Colorectal Cancer Refractory to Targeted Therapies, Chemotherapy and Immunotherapy
Articolo
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